This Biohack includes Hypermobile Ehlers-Danlos Syndrome, Vascular Ehlers-Danlos Syndrome, and Kyphoscoliotic Ehlers-Danlos Syndrome.
 
Now, through Epigenetic Biohacking, you can take back control of your health! Science has now shown that every dis-ease known has a specific gene that isn't functioning at optimal range. It's not necessarily the blueprint passed down to you from your parents, rather, in 90% of cases, it's caused by some epigenetic factor such as pathogens, heavy metals, or toxins, for example. The good news is that, because of epigenetics, which is how your environment and lifestyle influence the expression of your genes, you can now reverse the negative expression of your genes through diet, lifestyle, and targeted supplementation thereby decreasing symptoms, and kick-starting the self-healing process.
 
The Ehlers-Danlos Syndrome Biohack is a downloadable PDF file that's essentially a done-for-you remedy roadmap. We've done the research on the main genes and pathways involved with Ehlers-Danlos Syndrome, including Hypermobile Ehlers-Danlos Syndrome, Vascular Ehlers-Danlos Syndrome, and Kyphoscoliotic Ehlers-Danlos Syndrome, and show you exactly which natural compounds have the ability to modulate these genes and pathways back in your favor, thereby assisting the body in the reversal of symptoms and the dis-ease process itself.
 
Ehlers-Danlos syndrome is a group of inherited disorders that affect your connective tissues — primarily your skin, joints and blood vessel walls. Connective tissue is a complex mixture of proteins and other substances that provide strength and elasticity to the underlying structures in your body. 
 
Ehlers-Danlos Syndrome is a connective tissue disorder that is caused by defects in a protein called collagen that affect your connective tissues — primarily your skin, joints and blood vessel walls. Connective tissue is a complex mixture of proteins and other substances that provide strength and elasticity to the underlying structures in your body.
 
People who have Ehlers-Danlos syndrome usually have overly flexible joints and stretchy, fragile skin. This can become a problem if you have a wound that requires stitches, because the skin often isn't strong enough to hold them.
 
A more severe form of the disorder, called vascular Ehlers-Danlos syndrome, can cause the walls of your blood vessels, intestines or uterus to rupture. Because vascular Ehlers-Danlos syndrome can have serious potential complications. Vascular Ehlers-Danlos syndrome can weaken your heart's largest artery (aorta), as well as the arteries to other regions of your body. A rupture of any of these larger blood vessels can be fatal. The vascular type can also weaken the walls of the uterus or large intestines — which also may rupture.
 
Symptoms
 
There are many different types of Ehlers-Danlos syndrome, but the most common signs and symptoms include:
 
Overly flexible joints. Because the connective tissue that holds joints together is looser, your joints can move far past the normal range of motion. Joint pain and dislocations are common.
 
Stretchy skin. Weakened connective tissue allows your skin to stretch much more than usual. You may be able to pull a pinch of skin up away from your flesh, but it will snap right back into place when you let go. Your skin might also feel exceptionally soft and velvety.
 
Fragile skin. Damaged skin often doesn't heal well. For example, the stitches used to close a wound often will tear out and leave a gaping scar. These scars may look thin and crinkly.
 
Symptom severity can vary from person to person and depends on the specific type of Ehlers-Danlos syndrome that you have. The most common type is called hypermobile Ehlers-Danlos syndrome.
 
This Biohack includes natural remedies for the main genes and pathways associated with Hypermobile Ehlers-Danlos Syndrome, Vascular Ehlers-Danlos Syndrome, and Kyphoscoliotic Ehlers-Danlos Syndrome, including the recommended summary protocol for them.


EHLERS-DANLOS SYNDROME GENES & PATHWAYS:
 
COL1A1 Gene - Collagen Type I Alpha 1 Chain
PDGFRB Gene - Platelet Derived Growth Factor Receptor Beta
VIM Gene – Vimentin
AKT1 Gene - AKT Serine/Threonine Kinase 1
CSF2 Gene - Colony Stimulating Factor 2 (GM-CSF)
GRB2 Gene - Growth Factor Receptor Bound Protein 2 (Protein ASH)
PTPN11 Gene - Protein Tyrosine Phosphatase Non-Receptor Type 11 (SHP-2)
MAPK1 Gene - Mitogen-Activated Protein Kinase 1
SRC Gene - SRC Proto-Oncogene, Non-Receptor Tyrosine Kinase
HSPG2 Gene - Heparan Sulfate Proteoglycan 2
PILRA Gene - Paired Immunoglobin Like Type 2 Receptor Alpha
Focal Adhesion Kinase
ROCK Genes - Rho Associated Coiled-Coil Containing Protein Kinases
MYH10 Gene - Myosin Heavy Chain 10
Microtubule Associated Proteins
Actin Related Proteins
Signal Transduction SuperPath
PSMD14 Gene - Proteasome 26S Subunit, Non-ATPase 14
Integrin SuperPath
COL16A1 Gene - Collagen Type XVI Alpha 1 Chain
CHRD Gene – Chordin
Bone Morphogenetic Protein
Transforming Growth Factor Beta
Vascular Endothelial Growth Factor
Matrix Metallopeptidase (MMPs)
Human Neutrophil Elastase (ELANE)
Elastin
Elastase
FKBP14 Gene - FKBP Prolyl Isomerase 14
Unfolded Protein Response
Heat Shock Proteins
PLOD1 Gene - Procollagen-Lysine,2-Oxoglutarate 5-Dioxygenase 1
COL3A1 (Collagen Type III Alpha 1 Chain)
FBN1 Gene - Fibrillin 1
TNXB Gene - Tenascin XB
DCN Gene – Decorin
Glycosaminoglycan Metabolism